Seminar Psikologi Transpersonal

Seminar Psikologi Transpersonal.

Asesmen Pegawai

Asesmen Pegawai.

Proses Rekrutmen Karyawan

Proses Rekrutmen Karyawan.

Pelatihan Pembelajaran Bahasa Inggris Menggunakan Flash Card

Pelatihan Pembelajaran Bahasa Inggris Menggunakan Flash Card.

Pelatihan Psikologi Transpersonal Dalam Menjawab Realita Kehidupan

Pelatihan Psikologi Transpersonal Dalam Menjawab Realita Kehidupan.

Rabu, 27 April 2016

Neuropsychiatric Aspects of HIV Infection and AIDS
The human immunodeficiency virus (HIV) epidemic was identified in the 1980s and neurologists described several HIV-related central nervous system (CNS) syndromes within the first several years of the epidemic. Mental health professionals from nursing, social work, psychology, and psychiatry followed the plight of patients of the epidemic and helped to mobilize interest and galvanize a response. Initially, much of the work focused on grief and loss issues, as well as supportive psychotherapy, but quickly broadened to recognize a number of specific psychiatric conditions, including acquired immune deficiency syndrome (AIDS) dementia, the associated AIDS mania, increased rates of major depression, and psychiatric consequences of CNS injuries.
The first case of AIDS was reported in 1981. Analysis of specimens retained from persons who died before 1981, however, has shown that HIV infections were present as early as 1959. This suggests that in the 1960s and 1970s, HIV-related disorders and AIDS were increasingly common but unrecognized, particularly in Africa and North America. According to the Centers for Disease Control and Prevention (CDC), in 2005 almost 950,000 persons in Americas were diagnosed with full-blown AIDS since 1981. There were about 43,000 new infections, in 2004 with about 15,000 deaths. The CDC estimates that approximately 460,000 person are living with AIDS in the United States. The World Health Organization (WHO) estimates that, worldwide, 2.5 million adults and 1 million children have AIDS and about 30 million persons are infected with HIV. The CDC statistics on epidemiological data on AIDS in the United States is listed in Table 11-1.
Overview of HIV Transmission
Human immunodeficiency virus is a retrovirus related to the human T-cell leukemia viruses (HTLV) and to retroviruses that infect animals, including nonhuman primates. At least two types of HIV have been identified, HIV-1 and HIV-2. HIV-1 is the causative agent for most HIV-related diseases; HIV-2, however, seems to be causing an increasing number of infections in Africa. Other subtypes of HIV may exist, which are now classified as HIV-O. HIV is present in blood, semen, cervical and vaginal secretions, and, to a lesser extent, in saliva, tears, breast milk, and the cerebrospinal fluid of those who are infected. HIV is most often transmitted through sexual intercourse or the transfer of contaminated blood from one person to another. Unprotected anal and vaginal sex are the sexual activities most likely to transmit the virus. Oral sex has also been implicated, but rarely. Health providers should be aware of the guidelines for safe sexual practices and should advise their patients to practice safe sex (Table 11-2).
The chance of becoming infected after a single exposure to an HIV-infected person is relatively low: 0.8 to 3.2 percent for unprotected receptive anal intercourse, 0.05 to 0.15 percent with unprotected vaginal sex, 0.32 percent after puncture with an HIV-contaminated needle, and 0.67 percent after using a contaminated needle to inject drugs. The probability of transmission, however, could be higher, depending on the viral load of the contact person (which tends to be higher at the beginning and end of the course of the illness) or other factors, such as sexually transmitted diseases. The presence of sexually transmitted diseases, such as herpes or syphilis, or other lesions that compromise the integrity of skin or mucosa, further increases the risk of transmission. Transmission also occurs through exposure to contaminated needles, thus accounting for the high incidence of HIV infection among drug users. HIV is also transmitted by infusions of whole blood, plasma, and clotting factors, but not immune serum globulin or hepatitis B vaccine.
Although male-to-male transmission has been the most common route of sexual transmission in North America, male-to-female and female-to-male transmissions are increasing, and they represent most transmission worldwide. Some studies have shown that about 50 percent of the regular sex partners of persons with HIV infection become infected themselves, a statistic suggesting that some persons do not yet understand immunity or resistance to HIV infection.
Transmission by contaminated blood most often occurs when those abusing a substance intravenously (IV) share hypodermic needles without proper sterilization techniques. Transmission of HIV through blood transfusions, organ transplantation, and artificial insemination is no longer a problem now that donors are tested for HIV infection. Many hemophilia patients, however, received transfusions of HIV-infected blood products before HIV was identified as the causative agent. The risk of infection of health care workers after a needlestick is rare, about 1 in 300 incidents.
Children can be infected in utero or through breast-feeding when their mothers are infected with HIV. Zidovudine (Retrovir) and protease inhibitors taken by the HIV-infected pregnant woman prevent perinatal transmission in more than 95 percent of cases. Health workers are theoretically at risk because of potential contact with bodily fluids from HIV-infected patients. In practice, however, the incidence of such transmission is very low, and almost all reported cases have been traced to accidental punctures with contaminated hypodermic needles. No evidence has been found that HIV can be contracted through casual contact, such as by sharing a living space or a classroom with a person who is infected, although direct and indirect contact with an infected person's bodily fluids, such as blood and semen, should be avoided (Table 11-3).
After infection with HIV, AIDS is estimated to develop in 8 to 11 years, although this time is gradually increasing because
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of early treatment. Once a person is infected with HIV, the virus primarily targets T4 (helper) lymphocytes, also called CD4+ lymphocytes, to which the virus binds because a glycoprotein (gp120) on the viral surface has a high affinity for the CD4 receptor on T4 lymphocytes. After binding, the virus can inject its RNA into the infected lymphocyte, where the RNA is transcribed into DNA by the action of reverse transcriptase. The resultant DNA can then be incorporated into the host cell's genome and translated and eventually transcribed, once the lymphocyte is stimulated to divide. After viral proteins have been produced by lymphocytes, the various components of the virus assemble, and new mature viruses bud off from the host cell. Although the process of budding may cause lysis of the lymphocyte, other HIV pathophysiological mechanisms can gradually disable a patient's entire complement of T4 lymphocytes.
Table 11-1 The Centers for Disease Control and Prevention (CDC) Statistics on Epidemiological Data on Acquired Immunodeficiency Syndrome (AIDS) in the United States
  • 35% were white
  • 43% were black
  • 20% were Hispanic
  • 1% was of other race/ethnicity
Of the adults and adolescents with AIDS, 77% were men. Of these men,
  • 58% were men who had sex with men (MSM)
  • 21% were injection drug users (IDU)
  • 11% were exposed through heterosexual contact
  • 8% were both MSM and IDU.
Of the 93,566 adult and adolescent women with AIDS,
  • 64% were exposed through heterosexual contact
  • 34% were exposed through injection drug use
An estimated 3,927 children were living with AIDS at the end of 2004, of whom 97% probably acquired the infection from their mothers.
People with AIDS are surviving longer and are contributing to a steady increase in the number of people living with AIDS. This trend will continue as long as the number of new diagnoses exceeds the number of people dying each year.
Table 11-2 AIDS Safe-Sex Guidelines
Remember: Any activity that allows for the exchange of body fluids of one person through the mouth, anus, vagina, bloodstream, cuts, or sores of another person is considered unsafe at this time.
Safe-sex practices
Massage, hugging, body-to-body rubbing
Dry social kissing
Masturbation
Acting out sexual fantasies (that do not include any unsafe-sex practices)
Using vibrators or other instruments (provided they are not shared)
Low-risk sex practices
These activities are not considered completely safe:
French (wet) kissing (without mouth sores)
Mutual masturbation
Vaginal and anal intercourse while using a condom
Oral sex, male (fellatio), while using a condom
Oral sex, female (cunnilingus), while using a barrier
External contact with semen or urine, provided there are no breaks in the skin
Unsafe-sex practices
Vaginal or anal intercourse without a condom
Semen, urine, or feces in the mouth or the vagina
Unprotected oral sex (fellatio or cunnilingus)
Blood contact of any kind
Sharing sex instruments or needles
AIDS, acquired immunodeficiency syndrome.
(From Moffatt B, Spiegel J, Parrish S, Helquist M. AIDS: A Self-Care Manual. Santa Monica, CA: IBS Press; 1987:125, with permission.)
Table 11-3 Centers for Disease Control and Prevention (CDC) Guidelines for the Prevention of HIV Transmission from Infected to Uninfected Persons
Infected persons should be counseled to prevent the further transmission of HIV by:
  1. Informing prospective sex partners of their infection with HIV, so they can take appropriate precautions. Abstention from sexual activity with another person is one option that would eliminate any risk of sexually transmitted HIV infection.
  2. Protecting a partner during any sexual activity by taking appropriate precautions to prevent that person's coming into contact with the infected person's blood, semen, urine, feces, saliva, cervical secretions, or vaginal secretions. Although the efficacy of using condoms to prevent infections with HIV is still under study, the consistent use of condoms should reduce the transmission of HIV by preventing exposure to semen and infected lymphocytes.
  3. Informing previous sex partners and any persons with whom needles were shared of their potential exposure to HIV and encouraging them to seek counseling and testing.
  4. For IV drug abusers, enrolling or continuing in programs to eliminate the abuse of IV substances. Needles, other apparatus and drugs must never be shared.
  5. Never sharing toothbrushes, razors, or other items that could become contaminated with blood.
  6. Refraining from donating blood, plasma, body organs, other tissue, or semen.
  7. Avoiding pregnancy until more is known about the risks of transmitting HIV from the mother to the fetus or newborn.
  8. Cleaning and disinfecting surfaces on which blood or other body fluids have spilled, in accordance with previous recommendations.
  9. Informing physicians, dentists, and other appropriate health professionals of antibody status when seeking medical care, so that the patient can be appropriately evaluated.
HIV, human immunodeficiency virus; IV, intravenous.
(From MMWR Morb Mortal Wkly Rep. 1986;35:152, with permission.)
Diagnosis
Serum Testing
Techniques are now widely available to detect the presence of anti-HIV antibodies in human serum. The conventional test uses blood (time to result, 3 to 10 days) and the rapid test uses an oral swab (time to result, 20 minutes). Both tests are 99.9 percent sensitive and specific. Health care workers and their patients must understand that the presence of HIV antibodies indicates infection, not immunity to infection. Those with a positive finding on an HIV test have been exposed to the virus, have the virus within their bodies, have the potential to transmit the virus to another person, and will almost certainly eventually develop AIDS. Those with a negative HIV test result have either
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not been exposed to the HIV virus and are not infected or were exposed to the HIV virus but have not yet developed antibodies, a possibility if the exposure occurred less than a year before the testing. Seroconversion most commonly occurs 6 to 12 weeks after infection, although in rare cases seroconversion can take 6 to 12 months.
Table 11-4 Possible Indications for Human Immunodeficiency Virus (HIV) Testing
  1. Patients who belong to a high-risk group: (1) men who have had sex with another man since 1977; (2) intravenous drug abusers since 1977; (3) hemophiliacs and other patients who have received since 1977 blood or blood product transfusions not screened for HIV; (4) sexual partners of people from any of those groups; (5) sexual partners of people with known HIV exposure—people with cuts, wounds, sores, or needlesticks whose lesions have had direct contact with HIV-infected blood.
  2. Patients who request testing. Not all patients admit to the presence of risk factors (e.g., because of shame, fear).
  3. Patients with symptoms or acquired immunodeficiency syndrome (AIDS).
  4. Women belonging to a high-risk group who are planning pregnancy or who are pregnant.
  5. Blood, semen, or organ donors.
(Adapted from Rosse RB, Giese AA, Deutsch S, Morihisa JM. Laboratory and Diagnostic Testing in Psychiatry. Washington, DC: American Psychiatric Press; 1989:54, with permission.)
Counseling
The major issues in counseling persons about HIV serum testing are who should be tested; why a particular person should or should not be tested; what the test results signify; and what the implications are. Although specific groups of persons are at high risk for contracting HIV and should be tested (Table 11-4), any person who wants to be tested should probably be tested. The reasons for requesting a test should be ascertained to detect unspoken concerns and motivations that may merit psychotherapeutic intervention.
Past practices that may have put the testee at risk for HIV infection and safe sexual practices should be discussed (Table 11-5). During posttest counseling (Table 11-6), counselors should explain that a negative test finding implies that safe sexual behavior and the avoidance of shared hypodermic needles are recommended for the person to remain free of HIV infection. A positive test result indicates that the person is infected with HIV and can spread the disease. Those with positive results must receive counseling about safe practices and potential treatment options. They may need additional psychotherapeutic interventions if anxiety or depressive disorders develop after they discover that they are infected. Common issues and concerns are fear of disclosure, relationships with friends and family, employment and financial security, medical condition, and such psychological issues as self-esteem and self-blame. A person may react to a positive HIV test finding with a syndrome similar to posttraumatic stress disorder. Concern about minor physical symptoms, insomnia, and dependence on health care workers commonly arise. Adjustment disorder with anxiety or depressed mood may develop in as many as 25 percent of those informed of a positive HIV test result. Clinical interactions with patients should emphasize the meaning of a positive test result and should encourage reestablishment of emotional and functional stability.
Table 11-5 Pretest HIV Counseling
  1. Discuss meaning of a positive result and clarify distortions (e.g., the test detects exposure to the AIDS virus; it is not a test for AIDS).
  2. Discuss the meaning of a negative result (e.g., seroconversion requires time, recent high-risk behavior may require follow-up testing).
  3. Be available to discuss the patient's fears and concerns (unrealistic fears may require appropriate psychological intervention).
  4. Discuss why the test is necessary. (Not all patients will admit to high-risk behaviors.)
  5. Explore the patient's potential reactions to a positive result (e.g. â€Å“I'll kill myself if I'm positive”). Take appropriate necessary steps to intervene in a potentially catastrophic reaction.
  6. Explore past reactions to severe stresses.
  7. Discuss the confidentiality issues relevant to the testing situation (e.g., is it an anonymous or nonanonymous setting?). Inform the patient of other possible testing options where the counseling and testing can be done completely anonymously (e.g., where the result is not made a permanent part of a hospital chart). Discuss who has access to the test results.
  8. Discuss with the patient how being seropositive can potentially affect social status (e.g., health and life insurance coverage, employment, housing).
  9. Explore high-risk behaviors and recommend risk-reducing interventions.
  10. Document discussions in chart.
  11. Allow the patient time to ask questions.
HIV, human immunodeficiency virus; AIDS, acquired immunodeficiency syndrome.
(Reprinted with permission from Rosse RB, Giese AA, Deutsch SI, Morihisa JM. Laboratory and Diagnostic Testing in Psychiatry. Washington, DC: American Psychiatric Press; 1989:55, with permission.)
Table 11-6 Posttest HIV Counseling
  1. Interpretation of test result:
    Clarify distortion (e.g., â€Å“a negative test still means you could contract the virus at a future time; it does not mean you are immune from AIDS”).
    Ask questions about the patient's understanding and emotional reaction to the test result.
  2. Recommendations for prevention of transmission (careful discussion of high-risk behaviors and guidelines for prevention of transmission).
  3. Recommendations on the follow-up of sexual partners and needle contacts.
  4. If test result is positive, recommendations against donating blood, sperm, or organs and against sharing razors, toothbrushes, and anything else that may have blood on it.
  5. Referral for appropriate psychological support: HIV-positive patients often need access to a mental health team (assess need for inpatient versus outpatient care; consider individual or group supportive therapy). Common themes include the shock of the diagnosis, the fear of death, and social consequences, grief over potential losses, and dashed hopes for good news.
    Also look for depression, hopelessness, anger, frustration, guilt, and obsessional themes. Activate supports available to patient (e.g., family, friends, community services).
HIV, human immunodeficiency virus; AIDS, acquired immunodeficiency syndrome.
(From Rosse RB, Giese AA, Deutsch SI, Morihisa JM. Laboratory and Diagnostic Testing in Psychiatry. Washington, DC: American Psychiatric Press; 1989:58, with permission.)
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Couples who are considering taking the HIV antibody test must decide who will be tested and whether to go alone or together. The therapist should ask why they are considering taking the test; partners often for the first time discuss issues of commitment, honesty, and trust, such as sexual contacts outside the relationship. They need to be prepared for the possibility that one or both are infected and must discuss what effect this will have on their relationship.
Confidentiality
Confidentiality is a key issue in serum testing. No one should be given an HIV test without previous knowledge and consent, although various jurisdictions and organizations, such as the military, now require HIV testing for all inhabitants or members. The results of an HIV test can be shared with other members of a medical team, although the information should be provided to no one else except in the special circumstances discussed below. The patient should be advised against disclosing the results of HIV testing too readily to employers, friends, and family members; the information could result in discrimination in employment, housing, and insurance.
The major exception to restriction of disclosure is the need to notify potential and past sexual or IV substance use partners. Most patients who are HIV positive act responsibly. If, however, a treating physician knows that a patient who is HIV infected is putting another person at risk of becoming infected, the physician may try either to hospitalize the infected person involuntarily (to prevent danger to others) or to notify the potential victim. Clinicians should be aware of the laws about such issues, which vary among the states. These guidelines also apply to inpatient psychiatric wards when a patient who is HIV infected is believed to be sexually active with other patients.
Clinical Features
Nonneurological Factors
About 30 percent of persons infected with HIV experience a flulike syndrome 3 to 6 weeks after becoming infected; most never notice any symptoms immediately or shortly after their infection. When symptoms do appear, the flulike syndrome includes fever, myalgia, headaches, fatigue, gastrointestinal symptoms, and sometimes a rash. The syndrome may be accompanied by splenomegaly and lymphadenopathy. Rarely, acute aseptic meningitis develops shortly after infection, as does encephalopathy or Guillain-Barré syndrome.
In the United States, the median duration of the asymptomatic stages is 10 years, although nonspecific symptoms—lymphadenopathy, chronic diarrhea, weight loss, malaise, fatigue, fevers, night sweats—may variably appear. During the asymptomatic period, however, the T4 cell count almost always declines from normal values (>1,000/mm3) to grossly abnormal values (<200/mm3).
The most common infection in persons infected with HIV who have AIDS is Pneumocystis carinii pneumonia, which is characterized by a chronic, nonproductive cough, and dyspnea, sometimes sufficiently severe to result in hypoxemia and its resultant cognitive effects. Diagnosis is made with fiberoptic bronchoscopy and alveolar lavage. The pneumonia is usually treatable with trimethoprim and sulfamethoxazole (Bactrim, Septra) or pentamidine isethionate (Pentam), which can also be used for prophylaxis against the pneumonia. The other disease that was initially associated with the development of AIDS is Kaposi's sarcoma, a previously rare, blue-purple-tinted skin lesion. For unknown reasons, Kaposi's sarcoma is less commonly associated with cases of recently diagnosed AIDS.
Although Pneumocystis carinii pneumonia and Kaposi's sarcoma are the two classic AIDS-related infectious and neoplastic disorders, the severely disabled cellular immune system of patients infected with HIV permits the development of a staggering array of infections and neoplasms. The most common infections are from protozoa such as Toxoplasma gondii; fungi such as Cryptococcus neoformans and Candida albicans; bacteria such as Mycobacterium avium-intracellulare; and viruses such as cytomegalovirus and herpes simplex virus.
For psychiatrists, the importance of these nonneurological, nonpsychiatric complications lies in their biological effects on patients' brain functions (e.g., hypoxia with Pneumocystis carinii pneumonia) and their psychological effects on patients' moods and anxiety states. Further, because each of the conditions is usually treated by an additional drug, psychiatrists need to be aware of the adverse CNS effects of many medications.
Neurological Factors
An extensive array of disease processes can affect the brain of a patient infected with HIV (Table 11-7). The most important diseases for mental health workers to be aware of are HIV mild neurocognitive disorder and
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HIV-associated dementia. The latter is a cortical or subcortical type of dementia that can affect 50 percent of patients infected with HIV to some degree. Other diseases and complications of treatment must also be considered in the differential diagnosis of a patient who is HIV infected with neuropsychiatric symptoms. Symptoms such as photophobia, headache, stiff neck, motor weakness, sensory loss, and changes in level of consciousness should alert a mental health worker that the patient should be examined for possible development of a CNS opportunistic infection or a CNS neoplasm. HIV infection can also result in a variety of peripheral neuropathies that should prompt mental health clinicians to reconsider the extent of CNS involvement.
Table 11-7 Conditions Associated with Human Immunodeficiency Virus (HIV) Infection
Bacterial infections, multiple or recurrenta
Candidiasis of bronchi, trachea, or lungs
Candidiasis, esophageal
Cervical cancer, invasiveb
Coccidioidomycosis, disseminated or extrapulmonary
Cryptococcosis, extrapulmonary
Cryptosporidiosis, chronic intestinal (>1 month's duration)
Cytomegalovirus disease (other than liver, spleen, or nodes)
Cytomegalovirus retinitis (with loss of vision)
Encephalopathy, HIV-related
Herpes simplex, chronic ulcers (>1 month's duration); or bronchitis, pulmonitis, or esophagitis
Histoplasmosis, disseminated or extrapulmonary
Isosporiasis, chronic intestinal (>1 month's duration)
Kaposi's sarcoma
Lymphoid interstitial pneumonia and/or pulmonary lymphoid hyperplasiaa
Lymphoma, Burkitt's (or equivalent term)
Lymphoma, immunoblastic (or equivalent term)
Lymphoma, primary, of brain
Mycobacterium avium complex or M. kansasii, disseminated or extrapulmonary
Mycobacterium tuberculosis, any site (pulmonaryb or extrapulmonary)
Mycobacterium, other species or unidentified species, disseminated or extrapulmonary
Pneumocystis carinii pneumonia
Pneumonia, recurrentb
Progressive multifocal leukoencephalopathy
Salmonella septicemia, recurrent
Toxoplasmosis of brain
Wasting syndrome due to HIV
aChildren <13 years old.
bAdded in the 1993 expansion of the AIDS surveillance case definition for adolescents and adults.
(Adapted from 1993 revised classification system for HIV infection and expanded surveillance, case definition for AIDS among adolescents and adults. MMWR Morb Mortal Wkly Rep. 1992:41, with permission.)
Psychiatric Syndromes
HIV-Associated Dementia
The text revision of the fourth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV-TR) allows the diagnosis of dementia due to HIV disease in â€Å“the presence of a dementia that is judged to be the direct pathophysiological consequence of human immunodeficiency virus (HIV) disease.” (See Table 10.3-7.)
Although HIV-associated dementia is found in a large proportion of patients infected with HIV, other causes of dementia in these patients must be considered. These causes include CNS infections, CNS neoplasms, CNS abnormalities caused by systemic disorders and endocrinopathies, and adverse CNS responses to drugs. The development of dementia is generally a poor prognostic sign, and 50 to 75 percent of patients with dementia die within 6 months.
Mild Neurocognitive Disorder
A less severe form of brain involvement is called HIV-associated neurocognitive disorder, also known as HIV encephalopathy. It is characterized by impaired cognitive functioning and reduced mental activity that interferes with work, homemaking, or social functioning. No laboratory findings are specific to the disorder, and it occurs independently of depression and anxiety. Progression to HIV-associated dementia usually occurs but may be prevented by early treatment.
Delirium
Delirium can result from the same causes that lead to dementia in patients infected with HIV (Table 11-7). Clinicians have classified delirious states characterized by both increased and decreased activity. Delirium in patients infected with HIV is probably underdiagnosed, but it should always precipitate a medical workup of a patient infected with HIV to determine whether a new CNS-related process has begun.
Anxiety Disorders
Patients with HIV infection may have any of the anxiety disorders, but generalized anxiety disorder, posttraumatic stress disorder, and obsessive–compulsive disorder are particularly common.
Adjustment Disorder
Adjustment disorder with anxiety or depressed mood has been reported to occur in 5 to 20 percent of patients infected with HIV. The incidence of adjustment disorder in persons infected with HIV is higher than usual in some special populations, such as military recruits and prison inmates.
Depressive Disorders
A range of 4 to 40 percent of those infected with HIV have been reported to meet the diagnostic criteria for depressive disorders. The pre-HIV infection prevalence of depressive disorders may be higher than usual in some groups who are at risk for contracting HIV. Another reason for the reported variation in prevalence rates is the variable application of the diagnostic criteria; some of the criteria for depressive disorders (poor sleep and weight loss) can also be caused by the HIV infection itself. Depression is higher in women than in men.
Mania
Mood disorder with manic features, with or without hallucinations, delusions, or a disorder of thought process, can complicate any stage of HIV infection, but most commonly occurs in late-stage disease complicated by neurocognitive impairment.
Substance Abuse
Substance abuse is a problem both for IV substance abusers who contract HIV-related diseases and for other patients with HIV, who may have used illegal substances only occasionally in the past but may now be tempted to use them regularly to deal with depression or anxiety.
Suicide
Suicidal ideation and suicide attempts may increase in patients with HIV infection and AIDS. The risk factors for suicide among persons infected with HIV are having friends who died from AIDS, recent notification of HIV seropositivity, relapses, difficult social issues relating to homosexuality, inadequate social and financial support, and the presence of dementia or delirium.
Psychotic Disorder
Psychotic symptoms are usually later stage complications of HIV infection. They require immediate medical and neurological evaluation and often require management with antipsychotic medications.
Worried Well
The so-called worried well are those in high-risk groups who, although they are seronegative and disease free, are anxious about contracting the virus. Some are reassured by repeated negative serum test results, but others cannot be reassured. Their worried well status can progress quickly to generalized anxiety disorder, panic attacks, obsessive–compulsive disorder, and hypochondriasis.
Treatment
Prevention is the primary approach to HIV infection. Primary prevention involves protecting persons from getting the disease; secondary prevention involves modification of the disease's course. All persons with any risk of HIV infection should be informed about safe-sex practices and about the necessity to avoid sharing contaminated hypodermic needles. Preventive strategies, however, are complicated by the complex societal values surrounding sexual acts, sexual orientation, birth control, and substance abuse. Many public health officials have advocated condom distribution in schools and the distribution of clean needles to drug addicts. These issues remain controversial, although condom use has been shown to be a fairly (although not completely) safe and effective preventive strategy against HIV infection. Those who are conservative and religious argue that the educational message should be sexual abstinence. Many university laboratories and pharmaceutical companies are attempting to develop a vaccine to protect persons from infection by HIV. The development of such a vaccine, however, is probably at least a decade away.
The assessment of patients infected with HIV should include a complete sexual and substance-abuse history, a psychiatric history, and an evaluation of the support systems available to them. Clinicians must understand a patient's history with regard to sexual orientation and substance abuse, and the patient must feel that the therapist is not judging past or present behaviors. A therapist can often encourage a sense of trust and empathy in the patient by asking specific, well-informed, straightforward questions about the homosexual or substance-using culture. The therapist must also determine the patient's knowledge about HIV and AIDS.
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The homosexual community has provided a significant support system for those infected with HIV, particularly for persons who are gay and bisexual. Public education campaigns within this community have resulted in significant (more than 50 percent) reductions in the highest risk sexual practices, although some gay men still practice high-risk sex. Homosexual men are likely to practice safe sex if they know the safe-sex guidelines, have access to a support group, are in a steady relationship, and have a close relationship with a person with AIDS. Partly because of the many biases against them, IV substance users with AIDS have received little support, and little progress has been made in educating these persons who are a major reservoir for spread of the virus to women, heterosexual men, and children.
Pharmacotherapy
A growing list of agents that act at different points in viral replication has raised for the first time the hope that HIV might be permanently suppressed or actually eradicated from the body. At the time of this writing, the active agents were in two general classes: reverse transcriptase inhibitors and protease inhibitors. The reverse transcriptase inhibitors are further subdivided into the nucleoside reverse transcriptase inhibitor group and the nonnucleoside reverse transcriptase inhibitors. In addition to the new nucleoside reverse transcriptase inhibitors, nonnucleoside reverse transcriptase inhibitors, and protease inhibitors, other classes of drugs are under investigation. These include agents that interfere with HIV cell binding and fusion inhibitors (e.g., enfurvitide [Fuzeon]), the action of HIV integrase, and certain HIV genes such as gag, among others. Table 11-8 lists some of the available agents in each of these four categories.
The antiretroviral agents have many adverse effects. Of importance to psychiatrists is that protease inhibitors are metabolized by the hepatic cytochrome P450 oxidase system and, therefore, can increase levels of certain psychotropic drugs that are similarly metabolized. These include bupropion (Wellbutrin), meperidine (Demerol), various benzodiazepines, and selective serotonin reuptake inhibitors (SSRIs). Therefore, caution must be exercised in prescribing psychotropic drugs to persons taking protease inhibitors.
Table 11-8 Antiretroviral Agents
Generic Name Trade Name Usual Abbreviation
Nucleoside reverse transcriptase inhibitors
Zidovudine Retrovir AZT or ZDV
Didanosine Videx ddI
Zalcitabine Hivid ddC
Stavudine Zerit d4T
Lamivudine Epivir 3TC
Abacavir Ziagen
Nonnucleoside reverse transcriptase inhibitors
Nevirapine Viramune
Delavirdine Rescriptor
Efavirenz Sustiva
Protease inhibitors
Saquinavir Invirase
Ritonavir Norvir
Indinavir Crixivan
Nelfinavir Viracept
Fusion inhibitors
Enfurvitide Fuzeon
Beyond treatment directed specifically against HIV, many interventions are available to prevent and treat various complications of immunodeficiency caused by opportunistic viral, bacterial, fungal, and protozoan infections. Both survival and quality of life have improved substantially because of early diagnosis and treatment of these opportunistic conditions.
The use of combination antiretroviral regimens in conjunction with more specific treatments of complications has prolonged the survival of persons, both asymptomatic and symptomatic HIV infected. Despite progress in maintaining patients longer and in better states of health, the ultimate outcome, however, is still uncertain; that is, it is unclear at present whether any person who is HIV infected can expect to escape developing AIDS and ultimately dying. Those who are HIV infected are keenly aware of this prognosis, and their concern sometimes takes the form of psychiatric disturbances.
Novel treatments may also be useful. Neuronal excitotoxicity, mediated through the activation of glutamatergic receptors by the HIV envelope protein gp120, is a potentially important mechanism by which brain dysfunction might occur in HIV infection. Memantine is an open-channel antagonist of N-methyl-d-aspartate (NMDA)-type glutamate receptors that is generally well tolerated. It is currently being used as a treatment for dementia of the Alzheimer's type in Europe. On the assumption that an agent that could dislodge gp120 from neural receptor sites might be useful, an octapeptide called d-ala-peptide-t-amide (peptide t) has been used in phase II clinical trials. Compared with placebo, peptide t was associated with neuropsychological improvement in cognitively impaired individuals (with CD4 counts <200) and a reduced likelihood of progression of impairment on 6-month follow-up. Calcium channel inhibitors, which theoretically seemed potentially useful, have not proved successful.
The remaining forms of treatment are principally supportive. The most important step is to exclude other potentially treatable conditions, such as secondary infections or neoplasia, metabolic abnormalities with low-grade delirium, or other psychiatric disorders (e.g., major depressive disorder). Once the diagnosis is clear, then the usual supportive measures for neurocognitively impaired persons should be used. These include identifying areas of cognitive strength and deficit, reducing emphasis on areas that are now impaired (e.g., divided attention, speeded processing), emphasizing efforts to maintain good orientation and reality testing, and avoiding medications that might further compromise cognitive function, in particular, benzodiazepine drugs. If they must be used, such medications should be given at lower than usual doses. Antidepressant and antipsychotic agents, if indicated, may also have to be prescribed in much lower dosages (e.g., 25 percent of the usual recommended dosage).
Psychotherapy
Approaches
Major psychodynamic themes for patients infected with HIV involve self-blame, self-esteem, and issues regarding death. The psychiatrist can help patients deal with feelings of guilt regarding behaviors that contributed to infection or AIDS. Some patients with HIV and AIDS feel that they are being punished. Difficult health care decisions, such as whether to
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initiate or continue taking antiretroviral medication and terminal care and life-support systems, should be explored, and here denial of illness may be evident. Major practical themes involve employment, medical benefits, life insurance, career plans, dating and sex, and relationships with families and friends. The entire range of psychotherapeutic approaches may be appropriate for patients with HIV-related disorders. Both individual and group therapy can be effective. Individual therapy may be either short term or long term and may be supportive, cognitive, behavioral, or psychodynamic. Group therapy techniques can range from psychodynamic to completely supportive in nature.
Among the fears that must be confronted is the concern that once the individual's serostatus has been revealed, he or she has lost control of who next learns of the seroconversion. In deciding whether or not to tell others, patients must also address their sense of betrayal if they are not told. The same issues apply to the person's work environment. As a practical matter, the individual may need to decide whether to tell a trusted colleague in case of a job-related accident that might put others at risk of infection. Similarly, parents must decide when or whether to tell their children. Some parents want to tell very young children as soon as possible, whereas other parents prefer to withhold this information until their child's teenage years, for fear of â€Å“taking away their childhood.” The question of custody of children after the parent's death must be considered. The same question of timing will arise about when to tell children that they are seropositive. The parent must balance fears that telling the child's school will lead to discrimination while guarding their child's and others' safety in case of an accident.
The psychiatrist may have a special role regarding HIV treatment. The advent of protease inhibitors and the promise of additional increasingly effective therapies have brought hopes of a â€Å“cure” to patients and physicians alike. Even patients who have failed one or more rounds of combination therapies may find that family, friends, and physicians continue to be optimistic. The psychiatrist may be the only â€Å“safe” person to whom the patient can express discouragement, weariness, fear of treatment failure, and fury or guilt for not being able to tolerate successful therapy or for not responding to regimens that have benefited others. The psychiatrist also may be the only one confronting unrealistic expectations of cure or the assumption that safe sex practices are no longer relevant. Paradoxically, the therapeutic task also may be to examine the patient's reaction to a reprieve from certain death—the so-called second-life agenda.
Direct counseling regarding substance use and its potential adverse effects on health of the patient who is HIV infected is indicated. Specific treatments for particular substance-related disorders should be initiated if necessary for the total well-being of the patient.
Therapist-Related Issues
Countertransference issues and burnout of therapists who treat many patients infected with HIV must be evaluated regularly. Therapists must acknowledge to themselves their predetermined attitudes toward sexual orientation and substance use so that those attitudes do not interfere with the treatment of the patient. Issues regarding the therapist's own sexual identity, past behaviors, and eventual death may also give rise to countertransference issues. Psychotherapists who have practices with many patients infected with HIV can begin to have their effectiveness impaired by professional burnout. Some studies have found that seeing many such patients in a short time seems to be more stressful to therapists than seeing a smaller number of those infected with HIV over a longer period.
Involvement of Significant Others
The patient's family, lover, and close friends are often important allies in treatment. The patient's spouse or lover may have guilt feelings about possibly having infected the patient or may experience anger at the patient for possibly infecting him or her. The involvement of members of the patient's support group can help the therapist assess the patient's cognitive function and can also aid in planning financial and living arrangements for the patient. The patient's significant others may themselves benefit from the attention of the therapist in helping them cope with the illness and the impending loss of a friend or family member.
Partner Notification
Although no clear consensus has been reached, recommendations are that patients who are sexually active and infected with HIV should be counseled about potential risk to their sexual partners. Additionally, known partners should be notified of exposure risk and potential infection as well. Partner notification has been an extremely hotly debated topic; however, many states have developed legislation requiring or allowing either physicians or health department officials to notify partners of patients who are HIV infected of their risk. The current standard, despite the controversy, appears to be an obligation on the part of health care professionals to notify anyone who could be construed as clearly at risk and clearly identifiable and who may be unaware of their risk.
A particularly difficult situation is that of sex-industry workers known to be HIV infected and known to be working actively as prostitutes. Public health issues exist that pose a risk both for these patients and, depending on the politics of the circumstances, for their potential partners, clients, customers, victims, or victimizers. The response to this problem has ranged from a sense that sex-industry workers and their clients can make their own decisions and should be responsible for their own behavior all the way to the sentiment that such people should be arrested and jailed for attempted murder. It has additionally been noted that some sex-industry workers are impaired by a variety of psychiatric conditions, including cognitive impairment, major mental illness, personality disorder, and substance abuse disorders. These may further contribute to the sense that some sex-industry workers may be less than fully responsible for their behavior. Recommendations have been made for voluntary and involuntary interventions regarding these patients. Specific psychiatric interventions regarding competency, ability to consent, capacity, and, most importantly, treatment for the conditions that impair such people are critical to the mental health needs of patients with HIV.
References
Becker JT, Lopez OL, Dew MA, Aizenstein HJ. Prevalence of cognitive disorders differs as a function of age in HIV virus infection. AIDS. 2004;18[Suppl 1]: S11–S18.
Castellon SA, Hardy DJ, Hinkin CH, Satz P, Stenquist PK, van Gorp WG, Myers HF, Moore L. Components of depression in HIV-1 infection: Their differential relationship to neurocognitive performance. J Clin Exp Neuropsychol. 2006;28(3):420–437.
Davis HF, Skolasky RL Jr, Selnes OA, Burgess DM, McArthur JC. Assessing HIV-associated dementia: Modified HIV dementia scale versus the grooved pegboard. AIDS Reader 2002;12:29–31, 38.
Grant I, Atkinson JH Jr. Neuropsychiatric aspects of HIV infection and AIDS. In: Sadock BJ, Sadock VA, eds. Kaplan & Sadock's Comprehensive Textbook of Psychiatry. 7th ed. Vol. 2. Baltimore: Lippincott Williams & Wilkins; 2000:308.
Hilsabeck RC, Castellon SA, Hinkin CH. Neuropsychological aspects of coinfection with HIV and hepatitis C virus. Clin Infect Dis. 2005;41:S38–S44.
Maldonado JL, Fernandez F, Levy JK. Acquired immunodeficiency syndrome. In: Lauterbach EC, ed. Psychiatric Management in Neurological Disease. Washington, DC: American Psychiatric Press; 2000:271.
Martin L, Tummala R, Fernandez F. Psychiatric management of HIV infection and AIDS. Psychiatr Ann. 2002;32:133.
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Paul RH, Brickman AM, Navia B, Hinkin C, Malloy PF, Jefferson AL, Cohen RA, Tate DE, Flanigan TP. Apathy is associated with volume of the nucleus accumbens in patients infected with HIV. J Neuropsychiatry Clin Neurosci. 2005;17:167–171.
Paul RH, Flanigan TP, Tashima K, Cohen R, Lawrence J, Alt E, Tate D, Ritchie C, Hinkin C. Apathy correlates with cognitive function but not CD4 status in patients with human immunodeficiency virus. J Neuropsychiatry Clin Neurosci. 2005;17:114–118.
Pieper AA, Treisman GJ. Drug treatment of depression in HIV-positive patients: Safety considerations. Drug Saf. 2005;28(9):753–762.
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Treisman GJ, Angelino AF, Hsu J, Lyketsos CG. Neuropsychiatric aspects of HIV infection and AIDS. In: Sadock BJ, Sadock VA, eds. Kaplan & Sadock's Comprehensive Textbook of Psychiatry. 8th ed. Vol. 1. Baltimore: Lippincott Williams & Wilkins; 2005:426.
von Giesen HJ, Haslinger BA, Rohe S, Roller H, Arendt G. HIV Dementia scale and psychomotor slowing—The best methods in screening for neuro-AIDS. J Neuropsychiatry Clin Neurosci. 2005;17:185–191.

Materi Gangguan Kepribadian

NAMA MATA KULIAH  : PSIKOLOGI ABNORMAL
TOPIK    : GANGGUAN KEPRIBADIAN
PENGAMPU    : DRS. SUBANDI, MA            MEI 2000
________________________________________________________________

PENDAHULUAN:
Gangguan kepribadian secara garis besar dibagi menjadi 3 kelompok:
A. Gangguan kepribadian yang ditandai dengan tingkah laku aneh.  Termasuk di sini gangguan (1) kepribadian paranoid, (2)  Skizoid dan (3) Skizotipal.
B. Gangguan kepribadian yang ditandai dengan tingkah laku yang  berlebih-lebihan. Termasuk disini adalah gangguan kepribadian  (4) borderline, (5) histrionik, (6) narsisistik dan (7)  antisosial
C. gangguan kepribadian yang tungkah lakunya ditandai dengan  kecemasan dan ketakutan. Termasuk di dlamnya adalah gangguan  (8) kepribadian menghindar (avoidant), (9) tergantung  (dependant),  (10) obsesif-kompulsif dan (11) pasif-agerif. 

1. GANGGUAN KEPRIBADIAN PARANOID
Orang dengan gangguan kepribadian paranoid cenderung menginterpretasikan tingkah laku orang lain sebagai hal yang mengancam dirinya. Oleh karena itu mereka tidak bisa mempercayai orang lain. Mereka selalu menaruh rasa curiga. Mereka mempertanyakan kesetiaan dan ketulusan orang lain dalam hubungan sosial. Jangan-jangan informasi yang ada digunakan untuk menjebaknya. 
Orang paranoid merasa ada komplotan atau konspirasi yang berusaha menjelekkan, menyakiti atau menjatuhkan dirinya. 
Ciri-ciri gangguan kepribadian paranoid secara lebih jauh digambarkan dalam PPDGJ III, yaitu:
(a) Kepekaan yang berlebihan terhadap kegagalan dan penolakan
(b) kecenderungan untuk tetap menyimpan dendam, misalnya menolak  untuk memaafkan suatu penghinaan dan luka hati atau masalah  kecil
(c) kecurigaan dan kecenderungan yang mendalam untuk mendistorsikan  pengalaman dengan menyalah-artikan tindakan orang lain yang  netral atau bersahabat sebagai suatu sikap permusuhan atau  penghinaan.
(d) perasaan bermusuhan dan ngotot tentang hak pribadi tanpa  memperhatikan situasi yang ada
(e) kecurigaan yang berulang, tanpa dasar tentang kesetiaan seksual  dari pasangannya.
(f) kecenderungan untuk merasa dirinya penting secara berlebihan  yang bermanifestasi dalam sikap yang selalu merujuk  ke diri  sendiri
(g) preokupasi dengan penjelasan-penjelasan yang bersekongkol dan  tidak substantif dari suatu peristiwa, baik yang menyangkut  diri pasien sendiri maupun dunia pada umumnya.

2. GANGGUAN KEPRIBADIAN SKIZOID
Gambaran utama orang dengan kepribadian skizoid adalah bahwa mereka kurang mempunyai minat terhadap hubungan sosial. Mereka lebih suka menyendiri dan lebih suka memilih aktivitas yang dilakukan sendirian. Ada preokupasi dengan fantasi dan introspeksi yang berlebihan.  Oleh karena itu mereka hampir tidak punya teman dekat. Jika ada mungkin hanya satu atau dua. Memang mereka tidak ada keinginan untuk menjalin hubungan akrab. Meskipun mereka selalu menjaga jarak, tetapi mereka tetap memiliki kontak dengan realitas yang lebih baik dibandingkan dengan skizophrenia.  Emosi orang skizoid dangkal dan mendatar. Mereka jarang terlihat mengekspresikan kehangatan dan kelembutan atau kemarahan terhadap orang lain. Seakan mereka tidak perduli terhadap pujian dan kecaman.

3. GANGGUAN KEPRIBADIAN SKIZOTIPAL
Orang yang memiliki gangguan kepribadian skizotipal sering disebut sebagai suatu bentuk skizophrenia sederhana (simple schizophrenia). Mereka memiliki tingkah laku, cara berpikir, keyakian dan persepsi yang aneh.
Seperti pada skizoid, orang skizotipal tidak berminat terhadap hubungan sosial.  Mereka lebih suka menyendiri. Emosinya dangkal dan tumpul. Mereka kurang bisa menyambut ekspresi senyum orang lain. Tetapi kadang dia malah tersenyum dan tertawa sendiri ketika ada sesuatu  yang lucu dalam pikirannya. Orang skizoid memang sering tenggelam dalam fantasi dan pikirannya sendiri. Bahkan ketika bersama-sama dengan orang lain, misalnya dalam suatu kuliah atau pesta, tampak dia berbicara dengan dirinya sendiri. Mereka senang sekali berpikir hal-hal yang bersifat magis dan supernatural. Misalnya mereka yakin bahwa dirinya memiliki indera keenam. Tetapi mereka masih bisa mengontrol dirinya dan masih memiliki kontak dengan realitas.
4. GANGGUAN KEPRIBADIAN BORDERLINE
Istilah borderline personality sebenarnya digunanakn untuk menggambarkan orang yang tingkah lakunya beraada di ambang/perbatasan (border) antara neurosis dan psikosis. Mereka lebih parah dari neurosis tapi tidak separah psikosis, karena mereka masih memiliki kontak dengan realitas yang cukup baik. Tetapi tampaknya sekarang gangguan ini lebih dekat pada klasifikasi gangguan suasana perasaan hati (mood disorder). Mereka memiliki kemiripan dengan gangguan depresi dan bipolar.
Ciri pokok orang yang memiliki gangguan kepribadian Borderline adalah adanya kegagalan mengembangkan citra diri (self-image) yang stabil dan utuh. Hal ini diwujudkan dalam bentuk ketidak-pastian terhadap tujuan hidup, sehingga hidupnya seperti tidak punya arah. Karier atau pekerjaannya sering berubah secara mendadak. Mereka juga tidak memiliki kepastian terhadap nilia-nilai hidupnya. Kadang dia tampak memiliki moralitas tinggi, tapi kemudian berubah total.  Loyalitas dan hubungan interpersonalnya labil. Meskipun sebenarnya mereka tidak tahan sendirian. Perasaan terhadap orang lain bisa berubah dengan cepat. Mereka cepat sekali marah. Suatu saat teman dekatnya dianggap orang yang paling baik di dunia, saat lain jadi yang paling jelek. Ini karena dalam melihat orang lain, dia cenderung hitam - putih. Orang lain itu baik dalam segala hal atau jelek dalam segala hal. Oleh karena itu mereka tidak bisa menjalin hubungan dekat dengan orang lain dalam waktu yang cukup lama.
Alam perasaan orang borderline tidak menetap. Mereka mudah sekali berubah dari normal keadaan normal menjadi depresi atau cemas. Mereka juga mudah marah. Hal ini menyebabkan tingkah lakunya sulit diprediksi. Bahkan ada kecenderungan menyakiti atau mencederai diri sendiri. Apalagi ketika mereka dilanda perasaan  kosong dan bosan hidup yang memang sudah kronik.

5. GANGGUAN KEPRIBADIAN HISTRIONIK
Istilah histrionik berasal dari bahasa Yunani, histrio, yang artinya aktor, yang biasanya menjadi pusat perhatian orang. Orang histrionik memang selalu berusaha mencari perhatian orang dengan berbagai cara. Misalnya dengan menunjukkan ekspresi emosi yang berlebihan. Ketika dia sedang mengalami kesedihan sedikit saja sudah ditunjukkan dengan menangis terisak-isak.
Orang histrionik suka mendramatisasi hal-hal yang menimpa dirinya supaya orang lain menaruh perhatian. Hal yang sepele saja sering dibesar-besarkan. Misalnya kalau dia mengalami sakit sedikit saja, sudah menunjukkan sepertinya dia mengalami sakit berat.  Demikian juga dalam memberikan reaksi terhadap peristiwa yang terjadi di sekitarnya juga berlebihan.  Oleh karena itu mereka bisa jengkel dan marah secara tidak rasional.
 Hubungan interpersonal dengan orang lain meski tampak hangat dan menarik, tetapi sebenarnya hubungan itu dangkal saja. Mereka kurang bisa berempati dan cenderung egosentrik. Mereka hanya memperhatikan kepentingannya sendiri dan kurang mau memperhatikan kebutuhan orang lain. Bahkan terhadap orang lain mereka lebih banyak menuntut. Terutama berkaitan dengan keinginannya untuk dipuji dan diperhatikan. Kalau dia memakai baju baru dan orang lain tidak menghiraukannya, maka dia akan marah besar.
Orang-orang histrionik akan sangat tertarik sekali dengan profesi yang berkaitan dengan pemberian perhatian dan pemujaan, seperti aktris bintang film, foto model atau peragawan/wati. Tetapi meskipun di luar dia tampak sukses, banyak dipuji orang, ternyata mereka kurang bisa merasakan makna hidup.  

6. GANGGUAN KEPRIBADIAN NARSISISTIK
Istilah ini berasal dari nama seorang pemuda dalam mitologi Yunani, yaitu Narkisos, yang jatuh cinta dengan dirinya sendiri. Oleh karena itu orang yang memiliki gangguan kepribadian narsisistik memiliki kebanggan dan kecintan yang berlebihan tentang keunikan/kelebihan diri. Seperti pada orang histrionik, orang narsisistik juga membutuhkan perhatian dan pujian orang lain. Tetapi hal itu lebih di arahkan sebagai dukungan pada dirinya sendiri, sehingga dia lebih bangga dan cinta terhadap diri sendiri.
Untuk meningkatkan kebanggaAn dirinya, mereka memiliki preokupasi yang berlebihan dengan sukses, kekuasaan, cinta dan pasangan ideal. Atau ingin terkenal karena kecemerlangan pikiranatau karena kecantikan. Jika mereka mendapatkan hal itu, maka akan meningkatlah nilai dirinya di depan dirinya sendiri. 
Oleh karena itu seperti pada orang histrinik, orang narsisistik cenderung menyenangi pekerjaan yang membuat orang memujinya dan memperhatikannya, misalnya bintang film, foto model atau dalam dunia politik.
Orientasi yang berlebihan terhadap diri sendiri membuat mereka tidak memiliki empati terhadap orang lain dan menjadi sangat egoistik. Dalam berhubungan dengan orang lain mereka lebih banyak menuntut perlakuan istimewa dari pada memberikan sesuatu pada orang lain. Bahkan ada kecenderungan dia mengeksploitasi orang lain utnuk kepentingannya sendiri.

7. GANGGUAN KEPRIBADIAN ANTI SOSIAL (di bagian akhir tulisan ini)

8. GANGGUAN KEPRIBADIAN MENGHINDAR (AVOIDANT)
Orang dengan kepribadian menghindar sangat terganggu terhadap penolakan dan kritik orang lain, sehingga mereka tidak mau mengadakan hubungan sosial jika tidak yakin bahwa dia akan diterima. Di sini tampak bahwa sebenarnya orang dengan kepribadian menghindar tidak seperti orang skizoid yang tidak memiliki kehangatan hubungan sosial. Di sini mereka memiliki minat dan perasaan hangat untuk mendapatkan kasih sayang. Tetapi rasa takut ditolak membuat dia melakukan penarikan diri dari hubungan sosial. Mereka sangat takut dipermalukan dihadapan umum, sehingga mereka tak mau mengunjungi acara-acara berkumpul bersama tema-teman. Di sini mereka tampak memiliki rasa rendah diri. Mereka lebih suka  menjaga jarak dan mengambil peran perifer.  
Gangguan kepribadian menghindar memiliki beberpa kesamaan dengan orang yang mengalami soisal phobia. Keduanya takut ditolak dan dikritik orang lain. Tetapi dalam phobia sosial orang takut pada situasi bersama yang spesifik. Misalnya dalam pesta atau ketika diminta berbicara di depan umum. Pada orang dengan kepribadian menghindar cenderung menghindari hubungan interpersonal. Menurut Mark (dalam Rathus, 1983) kepribadian menghindar tidak bisa menjaga hubungan sosial untuk jangka yang cukup lama karena pada umumnya mereka tidak memiliki ketrampilan sosial (social skill)

9. GANGGUAN KEPRIBADIAN DEPENDANT
Ciri utama orang yang memiliki gangguan kepribadian dependant adalah bahwa mereka sangat sulit melakukan suatu perbuatan yang dipilihnya sendiri. Mereka selalu berusaha mencari nasehat dan dukungan orang lain untuk melakukan perbuatan kecil sekalipun. Misalnya apakah mau pergi ke luar kota dengan naik bis atau naik kereta api. Pada anak-anak dan remaja mereka akan meminta orangtuanya untuk memilihkan pakaian, memilihkan makanan atau mencari sekolah. Pada orang dewasa mereka akan meminta orang lain untuk memilihkan karier yang cocok untuknya. Bahkan kadang mereka minta tolong untuk memilihkan pasangan hidupnya. Jika merke sudah menikah, mereka akan menggantungkan diri pada pasangan hidupnya dalam memilih untuk melakukan sesuatu mislnya kemana akan pergi ke rekreasi atau bagaimana mengatur keuangan. 
Perilaku yang pasif seperti di atas tampaknya dilandasi oleh perasaan tidak berani bertanggung jawab. Mereka akan menghindari peran-peran sosial di mana mereka harus banyak memikul tanggung jawab. Mereka cenderung memilih posisi sebagai bawahan yang hanya mengikuti perintah atasannya.  Mereka memang sangat memperhatikan kebutuhan orang lain yang digantungi itu. bahkan kadang tidak memperhatikan kepentingan dirinya sendiri supaya dia tetap mendapatkan tempat bergantung. Mereka sangat penurut dan suka mengalah, sehingga kadang banyak dimanfaatkan oleh orang lain.
Mereka kurang percaya pada kemampuan diri (merasa tak mampu, merasa bodoh dsb.), sehingga mereka umumnya memiliki prestasi yang jauh di bawah kemampuan yang sebenarnya mereka miliki. Mereka tidak suka dengan persaingan. Lebih menyukai ketenangan dan ketenteraman.

10. GANGGUAN KEPRIBADIAN OBSESSIVE KOMPULSIVE
Ciri utama orang yang memiliki gangguan obsesif kompulsif adalah adanya kecenderungan perfeksionistis. Ingin melakukan sesuatu dengan benar dan takut membuat kesalahan dalam melakukan suatu pekerjaan. Mereka sangat memperhatikan hal-hal kecil dan detail. Preokupasi dengan aturan, urutan, jadwal dan daftar. 
Aturan bagi orang yang memiliki gangguan obsesif kompulsif menjadi teramat penting untuk diperhatikan. Mereka menjadi orang yang sangat disiplin. Tidak pernah datang terlambat ke sekolah atau ke tempat kerja atau melakukan kegiatan sesuai dengan jadwal. Mereka juga akan mengatur rumahnya dengan rapih dan bersih. Jika ada hal yang tidak beres sedikit membuatnya jengkel.  Jadwal kegiatannya  sehari-hari telah tersusun dalam daftar yang rapi. Mereka mempunyai preokupasi terhadap pekerjaan. Bahkan ada kecenderungan lebih mementingkan untuk berkarya dan karier daripada menjalin persahabatan.
   Hubungan sosial ornag obsesif kompulsif memang menjadi sangat kaku karena segala seuatu telah diatur secara rapi. Mereka tidak memiliki dan tidak mampu mengekspresikan  kehangatan dan kelembutan perasaan. Selanjutnya hal ini akan mempengaruhi kemampuannya berempati yang mnjadi rendah. Dia tidak bisa mersakan apa yang dirasakan orang lain. Atau memang dia tidak mau perduli dengan perasaan orang lain. Yang lebih mereka perhatikan dalam hubungan sosial adalah aturan dan tata krama. Mereka selalu menjaga formalitas setiap saat, sehingga tampak selalu serius.
Dalam pergaulan mereka berpegang teguh pada hierarki sosial. Orang yang lebih rendah kedudukannya dari dia tidak selayaknya duduk sejajar dengannya. Dalam situasi ini dia akan merasa superior dan cenderung bersikap otoriter. Mereka mendesak orang lain melaksanakan pekerjaan sesuai dengan caranya.
11. GANGGUAN KEPRIBADIAN PASIF AGRESIF
Orang dengan kepribadian pasif-agresif tidak mengekspersikan agresifitas dengan orang lain secara terbuka, tetapi dengan cara-cara yang pasif. Untuk menunjukkan permusuhan dan kejengkelannya dia bukannya marah-marah ataupun memukul orang yang menjengkelkannya, tetapi justru sebaliknya dia akan menjadi diam, pura-pura tidak mendengar jika disuruh, sengaja melakukan kesalahan sengaja "lupa". Misalnya seorang suami yang merasa jengkel karena disuruh membantu istrinya mencuci piring, tidak menyatakan terus ternag kejengkelannya itu tetapi ketika disuruh dia pura-pura tidak mendengar sampai istrinya jengkel. Kemudian ketika dia terpaksa mencuci piring untuk membalas kejengkelan itu dia kemudian memecahkan piring dengan sengaja.
Penampilan luar orang dengan kepribadian pasif agresif adalah keras kepala dan bandel. Dalam bekerja sering berlambat-lambat atau tidak efisein dengan sengaja. Dalam berhubungan sosial mereka sulit diajak kerja sama. Selalu melawan dan bersikap negativistik. Suka mencari kesalahan orang lain dan mengkritiknya. Tetapi dia sendiri merasa jengkel jika dikritik. Lebih banyak mengutarakan keluhan-keluhan kepada orang lain.

7.  GANGGUAN KEPRIBADIAN ANTI SOSIAL
Istilah yang pertama kali digunakan untuk menyebut gangguan kepribadian ini adalah 'Psikopat'. Sebagai klasifikasi psikiatris konsep psikopat bermula dari label yang diberikan Philip Phinel kepada pasiennya yang menderita: "manie san delire". Pasien ini berasal dari keluarga bangsawan yang kaya. Meskipun demikian, dia tidak pernah puas terhadap semua keinginannya, mudah marah dan melakukan tindakan agresip.
Label lain diberikan oleh Pritchard yaitu 'moral insanity'. Sedangkan Rush menyebutnya sebagai 'moral dearangement'. Konsep yang lebih profesional berawal dari anggapan bahwa tingkah laku ini bersifat herediter. Kemudian Koch mengusulkan istilah 'Psychopatie inferiority'. Tetapi ternyata bahwa orang psikopat ini tidak mempunyai gangguan fisik maupun psikopatologis seperti yang dikenal, misalnya: neurosis, psikosis maupun retardasi mental, maka beberapa ahli berpendapat bahwa sebenarnya mereka bukan sakit dalam arti fisik dan psikologis, tetapi 'sakit' dalam pengertian sosial, sehingga timbullah istilah 'Sosiopat'. (Ullmann and Krasner, 1969).
Istilah yang sekarang banyak digunakan adalah 'anti sosial'. PPDGJ II juga menggunakan istilah ini, dan menggolongkan dalam kategori gangguan kepribadian dengan kode 301.7.

PENGERTIAN
Dibanding dengan jenis keabnormalan yang lain, gangguan kepribadian psikopat ini agak sulit untuk dikenal. Sebab seorang psikopat tidak menunjukkan gejala yang jelas seperti pada neurosis, retardasi mental ataupun psikosa. Buss mengatakan bahwa gejala pokok psikopat adalah ketidakmampuan mereka mengendalikan diri, sehingga menimbulkan sikap yang asosial dan immature. Ini sangat berlawanan dengan orang neurotis yang mempunyai kontrol diri berlebihan. Jadi jika orang normal mempunyai kpntrol diri yang cukup baik, dan neurotis mempunyai kontrol yang berlebihan, maka psikopat hampir tak ada kontrol sama sekali (Siti Meichati,1970).
Menurut Eysenk dan Mcili (1972) psikopat adalah orang yang impulsip, tak bertanggungjawab, hedonistik dan memiliki kepribadian ganda yang tak mampu menghayati hubungan interpersonal. Mereka tak punya rasa bersalah, rasa penyesalan, empati, afeksi serta tak ada perhatian sungguh-sungguh untuk kebaikan orang lain. Meskipun dia pandai berkata tentang hal-hal yang emosional, tetapi semuanya hanya dibuat-buat saja.
Demikian juga Maslow dan Mittleman (1951) mengatakan bahwa istilah psikopat menunjuk kepada suatu kondisi dimana seseorang mengalami kesulitan menghayati hubungan interpersonal, hubungan sosial maupun menghayati nilai-nilai moral. Coleman (1976) menambahkan bahwa semua ini menyebabkan psikopat senantiasa berkonflik dengan masyarakat.
Jadi psikopat atau kepribadian anti sosial adalah suatu gangguan kepribadian yang membuat seseorang tidak mempunyai kemampuan mengendalikan diri dan menghayati kehidupan emosional, sehingga menimbulkan tindakan yang antisosial.

KARAKTERISTIK
Coleman (1976) memberikan gambaran yang lebih jelas tentang kepribadian antisosial:    
1. Perkembangan hati nurani yang tidak baik dan kurang memiliki  rasa bersalah.
Mereka tidak memahami dan menghayati nilai-nilai moral dan etika secara sungguh-sungguh. Mungkin saja dia pandai berbicara tentang masalah moral dan keagamaan, tetapi semuanya hanya berifat verbalistik, hanya omongan saja. Demikian juga kalau dia melakukan kesalahan atau tindakan agresip, mungkin saja dia pandai meyakinkan bahwa dia sudah merasa bersalah dan menyesal, tapi di lain waktu juga melakukan lagi, sebab pada dasar mereka tidak punya rasa bersalah, berdosa atau penyesalan yang dalam.

2. Tingkah lakunya impulsip dan tak bertanggungjawab. Toleransi  frustasinya rendah.
Kepribadian psikopat sama sekali tidak masak. Seperti seorang kanak-kanak, mereka tak punya kontrol diri. Mereka selalu ingin bersenang-senang. Semua keinginan harus terpenuhi, maunya menang sendiri. Sikapnya sangat Egosentris, tak pernah mempedulikan hak-hak dan kepentingan orang lain. Kalau keinginannya tidak terpenuhi maka mudah timbul frustasi. Mereka juga tak mampu bertanggungjawab atas segala tindakannya. Amat mudah bagi mereka melakukan tindakan yang melanggar hukum, tanpa mempedulikan akibatnya.
3. Pandai bermuka manis untuk menekan dan memanfaatkan orang lain  demi kepentingan sendiri.
Memang benar bahwa orang psikopat itu biasanya mempunyai intelegensi yang tinggi. Penampilannya sangat meyakinkan, pandai bermuka manis, pandai berbicara dan merayu, tapi semua itu digunakan untuk menipu orang lain, demi tercapainya keinginan sendiri. Mereka terkenal sebagai penipu dan pendusta yang patologis dan lihai. Sangat pandai memutarbalikkan fakta dan membuat berbagai macam alasan, sehingga terhindanr dari kesalahan.
4. Menolak otorita dan tak mampu mengambil pelajaran dari  pengalaman.
Mereka ingin bebas dan tak mau diatur. Bahkan aturan-aturan dan norma dalam masyarakat itu seakan-akan tak ada, sehingga melakukan pealnggaran hukum itu suatu hal yang biasa. Jadi kalau mereka melakukan tindakan kriminal itu sifatnya bukan profesional, karena bagi mereka itu hal yang biasa. Dan kalaupun diberi hukuman berkali-kali mereka tetap melakukannya. Mereka tak bisa mengambil pelajaran dari tindakannya di masa lalu maupun memikirkan akibat tindakannya di masa mendatang. Yang penting adalah hari ini dan di sini 'saya' senang.
5. Tidak mampu membina hubungan interpersonal yang baik.
Karena sifatnya yang egosentris, orang psikopat tak bisa menenggang rasa pada orang lain. Mereka tak bisa merasakan cinta, kasih sayang, simpati, persahabatan, kesetiaan. Orang lain hanya dianggap sebagai obyek untuk memuaskan keinginan-keinginannya saja.

Benyamin Kleinmuntz (1980) secara lebih jelas dan sistematis menyebutkan beberapa karkteristik kepribadian psikopat:
 1. Tidak mampu membentuk hubungan yang penuh loyalitas, dan  memperlakukan manusia seperti benda.
 2. Tidak mempunyai rasa bersalah (guilty feeling), penyesalan yang  dalam tapi pandai meyakinkan orang lain dengan omongannya  bahwa dia betul-betul merasa bersalah.
 3. Tidak mampu mengambil pelajaran dari pengalaman, dan berulang- ulang melakukan tindakan kriminal.
 4. Cenderung menuruti keinginan dan mencari kesenangan saja.
 5. Sikapnya impulsip dan kekanak-kanakan.
 6. Agresifitasnya tinggi, cenderung destruktif jika frustasi.
 7. Penampilan sangat meyakinkan dan inteligen, tapi hanya  superfisial saja.
 8. Tidak mampu bertanggungjawab.
 9. Pembohong yang patologis.
10. Miskin perasaan, tidak bisa menghayati emosi.
11. Egosentris.
12. Kurang memiliki insigtt, tidak mampu melihat diri sendiri seperti yang dilihat orang lain.
13. Tingkah laku seksualnya ganas, tak bisa merasakan cinta.

TEORI-TEORI PSIKOPAT (ANTI-SOSIAL) 
Ada beberapa teori yang berusaha mengungkap timbulnya kepribadian antisosial/psikopat, baik yang bersifat biologis, psikologis, maupun sosiokultural.

1. Teori Biologis:
Termasuk dalam teori ini adalah adanya anggapan bahwa kepribadian psikopat bersifat herediter, suatu bentuk kelainan yang diturunkan dan dibawa sejak lahir. Tetapi menurut penelitian Mc Crod & Mc Crod dapat disimpulkan bahwa faktor genetik & konstitusional dari psikopat ternyata tidak cukup meyakinkan. Sehingga hubungan antara hereditas dan psikopat tak dapat dipertahankan (Ullman & Krasner, 1969).
Pandangan yang bersifat biologis lainnya timbul setelah ditemukan adanya kelainan getaran otak pada psikopat. Ternyata hasil EEG mereka menunjukkan keabnormalan, yakni pada bagian lobus temporalis, gelombang otaknya sangat rendah. Hare menyimpulkan  bahwa kelainan semacam itu menunjukkan tidak berfungsinya mekanisme pertahanan pada susunan saraf sentral, sehingga membuat orang itu mengalami kesulitan dalam belajar mengontrol tingkah lakunya (Coleman, 1976). Tetapi hasil penelitian yang lain menyatakan bahwa tidak semua psikopat mengalami kelainan EEG, sebagian juga normal.

2. Teori Interpersonal:
Teori ini meletakkan titik pangkal gangguan kepribadian psikopat pada hubungan interpersonal, terutama hubungan antara ibu dan anak pada masa kanak-kanak.
Pandangan psikoanalisa menganggap bahwa psikopat muncul karena adanya kesalahan dalam perkembangan super Ego. Aspek pengontrol perilaku manusia ini tidak timbul dengan baik. Demikian juga Ego tidak berkembang sebagaimana layaknya. Id yang paling berkuasa. Baik Ego maupun Super Ego tunduk mengikuti kemauan Id (Siti Meichati, 1970).

Berdasarkan pada penelitian empiris, Coleman (1956 dan 1976) menyebutkan beberapa bentuk hubungan dalam keluarga, yang bisa menimbulkan kepribadian psikopatik:
a). Kehilangan orangtua di masa kanak-kanak dan adanya deprivosi  emosional.
Sejumlah penelitian menemukan bahwa sebagian besar psikopat mempunyai pengalaman traumatik di masa kanak-kanaknya, karena ditinggal orangtuanya atau kedua orangtuanya bercerai. Tetapi menurut Hare bukan kehilangan orangtua itu yang menjadi kunci persoalannya, melainkan karena gangguan-gangguan emosional (emotional turbances) dengan adanya hubungan antara anggota keluarga yang patogenik.

b) Penolakan orangtua atau orangtua yang tidak konsisten.
Menurut Mc Cord dan Mc Cord, penolakan orang tua yang berat dan kurangnya afeksi orangtua merupakan penyebab utama timbulnya psikopat. Selanjutnya Buss mengemukakan dua tipe perilaku orangtua yang menyertai timbulnya kepribadian ini, yaitu :
Pertama, orangtua bersikap dingin dan hubungan dengan anak terlalu jauh. Tidak ada kehangatan emosional, cinta dan kasih sayang. Hal ini akan tetap dibawa oleh anak pada masa dewasanya, sehingga dia pun bersikap dingin terhadap orang lain, tak bisa berempati atau terlibat secara emosional.
Kedua adalah tipe perilaku orangtua yang tidak konsisten baik dalam memberikan afeksi maupun dalam memberikan hadiah dan hukuman. Biasanya orangtua itu sendiri tidak konsisten terhadap peraturan yang dibuatnya. Misalnya anak dilarang berbohong, tetapi orangtua sendiri tak dapat dipercaya omongannya. Hal ini membuat anak kehilangan model yang mantap untuk mengadakan imitasi dan gagal mengembangkan identitas diri yang jelas. Demikian juga kalau orangtua tidak konsisten dalam memberikan hadiah dan hukuman. Misalnya anak mencuri uang, mestinya harus dihukum tetapi ternyata ibu mudah merasa iba setelah anak menyatakan penyesalan (meskipun hanya pura-pura) sehingga tak tega menghukumnya. Hal ini terjadi berulangkali. Dari sini anak belajar, bahwa dengan pernyataan sesal saja dia sudah lepas dari hukuman. Disiplin yang tidak konsisten ini akan menimbulkan rendahnya perkembangan hati nurani.

c) Model orangtua dan interaksi dalam keluarga yang salah.
GREEACRE melaporkan hasil penelitian terhadap psikopat dari keluarga kelas menengah. Ternyata kebanyakan ayahnya adalah orang yang sukses dan terpandang di masyarakat tetapi dingin dan menaruh jarak pada anaknya. Sedangkan ibunya pemalas dan pecinta kenikmatan. Sebenarnya keluarga ini penuh dengan konflik dan skandal, tapi mereka tetap mempertahankan penampilan di depan masyarakat sebagai keluarga yang harmonis. Dengan demikian anak mempelajari bahwa yang penting itu adalah penampilan bukan kenyataan yang sebenarnya. Dia juga mempunyai sikap dingin seperti ayahnya.


3. Teori Sosio-kultural:
Beberapa kondisi sosiokultural dalam masyarakat ternyata juga dapat mengakibatkan timbulnya psikopat. Terutama masyarakat yang norma dan aturan-aturan hidupnya sudah berantakan, tidak teratur, dan masanya terasing dari masyarakat lebih luas bahkan bersikap bermusuhan degan mereka. Ini adalah tempat yang subur bagi timbulnya orang-orang yang hati nuraninya tidak berkembang dengan baik, yang kurang perhatian pada orang lain dan cenderung bertingkah laku destruktif.

TREATMENT
Karena memang individu dengan kepribadian psikopat ini bukan termasuk dalam kategori psikopatologi seperti retardasi mental, neurosis ataupun psikosis, maka mereka jarang mendapat perhatian dari rumah sakit jiwa. Mereka yang nyata melanggar hukum, biasanya dimasukkan dalam program-program rehabilitasi di lembaga-lembaga tertentu, tapi sejauh ini belum efektif.
Kleinmuntz (1980) menyebutkan beberapa treatment yang pernah dicobakan pada psikopat:
1. Psikoterapi tradisional:
Tehnik psikoterapi tradisional dengan memberikan konsultasi ternyata gagal karena mereka pandai berpura-pura, kurang mempunyai insight, impulsip, sikapnya meremehkan dan kurang motivasi terhadap treatment.
2. Somatic Treatment:
Ini dilakukan  berdasar pada teori biologis yang telah disebutkan, yaitu dengan memberikan shock-therapy secara berkala. Ada pasien yang tingkah laku maupun EEGnya menjadi baik, tapi ada juga yang EEGnya justru bertambah buruk. Tehnik ini masih meragukan.
3. Pendekatan Farmakologis:
Yaitu dengan memberikan obat-obatan pada para psikopat. Tetapi hasilnya juga masih dipertanyakan. Demikian juga pembedahan otak, ternyata tidak efektif.
4. Modifikasi Perilaku:
Tehnik ini dilaksanakan dengan memberikan hadiah pada tingkah laku prososial dan memberikan hukuman pada tingkah laku antisosial. Meskipun tampaknya tehnik ini sedikit memberi harapan untuk memperbaiki psikopat, tapi hanya diterapkan untuk mengubah tingkah lakkkku delinquent, dan tidak pada kepribadian antisosial itu sendiri.


DAFTAR PUSTAKA 
Coleman, J.C. Abnormal Psychology and Modern Life, 5th
Edition, Taraporevala Sons & Co, Private Ltd, India
1956

Kleinmuntz, B. Essential of Abnormal Psychology. Second
Edition. Harper & Row Publishers, San Fransisco.
1980

Maslim, R. 1996. Buku Saku Pedoman Penggolongan Diagnosis Gangguan  Jiwa. Jakarta: 

Rathus, S.A. & Nevid, J.S. 1991. Abnormal Psychology. Engelwood  Cliff, New Jersey: Prentice Hall.

Siti Meichati. Psikologi Abnormal dan Psikopatologi. Fa
kultas Psikologi UGM, Yogyakarta. 1970

Ullmann & Krasner. A Psychology Approach to Abnormal Be
havior. Prentice-Hall, Inc, Engelwood Cliffs, New 
Jersey. 1969.     


KASUS-KASUS
Suatu hari Tina, seorang siswa SMA, sedang makan sambil nonton film Knigh Rider di TV. Ibunya yang sedang didapur memanggil-manggil dan menyuruhnya membantu mencuci piring. Tapi Tina pura-pura tidak mendengar. Ia tetap asyik dengan makanan dan totntonannya. Berkali-kali ibunya memanggil, tapi tidak ada jawaban. Karena jengkel, ibunya menghampiri Tina.
"Apa kamu tidak punya kuping?" kata ibunya jengkel.
Tina diam saja pura-pura tidak tahu.
"Mama kan dari tadi memanggil kamu. Kamu kok diam saja kayak anak bego."
"Kan Tina baru makan dan nonton TV, Ma," Tina membikin alasan.
"Habis makan nanti bantu Mama cuci piring. Ngerti?"
Tina diam saja.
"Ngerti, nggak?" Ibunya tambah jengkel.
"Ya......ngerti, Ma," jawab Tina malas.
Ketika ibunya pergi ke dapur, Tina asyik kembali dengan makanan dan Knigh Rider-nya seakan tidak ada apa-apa sebelumnya. Dengan sengaja ia mengulur-ulur waktu, supaya kalau dia sudah selesai makan, ibunya juga selesai mencuci piring.
Ibunya memanggil dari dapur sekali lagi.
Tina diam seribu bahasa.
  
******


Ketika Heru, seorang mahasiswa Psikologi, akan mencari tempat kost baru ia mengetuk pintu kamar yang ada di situ. Beberapa kali diketuk pintu tak ada jawaban. Padahal Heru terdengar suara radio berbunyi dari luar, pasti ada orangnya di dalam. 
Beberapa saat kemudian seraut wajah laki-laki muncul dengan menyibakkan sedikit korden jendela. Heru agak terkejut melihat wajah yang agak menakutkan itu. Sorot matanya tajam penuh selidik.
"Mau cari siapa?" tanya orang itu dengan penuh kecurigaan.
"Mau cari kost. Apa masih ada kamar kosong?"tanya Heru.
"Itu tempat ibu kost-nya. Tanya sendiri ke sana," kata orang itu tetap dari balik korden jendela.
Heru menjadi terkesan dengan penghuni kamar itu. Ketika dia sudah beberapa hari pindah di tempat kost itu, dia mencoba mengadakan pendekatan. Dengan masih menaruh kecurigaan orang itu mempersilahkan Heru bertamu ke kamarnya. Heru agak heran melihat ada sebuah kayu besar dibalik pintu.
"Untuk apa kayu ini?" tanya Heru 
"Banyak orang jahat di sekitar ini." kata penghuni kamr itu.  Heru hanya tersenyum saja melihat keanehan perilaku tteman barunya.


Tomo adalah seorang eksekutif muda yang sukses. Ia sangat serius dengan kariernya. Baginya karier adalah yang paling utama dalam hidupnya, sehingga meskipun dia sudah berumur 35 tahun, masih tetap sendirian.
Ia dikenal bawahannya sebagai pimpinan yang sangat disiplin dengan waktu. Jam 8 pagi tepat sudah sampai di kantor dan pulang ke rumah tepat jam 5 sore tepat. Seluruh kegiatannya sudah terjadwal dengan ketat dan dia selalu tepat waktu. Kalau mengerjakan suatu pekerjaan harus sesuai dengan urutannya. Demikian juga ia selalu menekankan kepada bawahannya supaya seperti dia dalam melakukan pekerjaan
Kalau orang masuk ke dalam kantornya akan terkesan dengan kerapihannya. Segala sesuatu diatur. Tak ada satu barangpun yang terkesan semrawut. Berkas-berkas yang ada di atas mejanya tertumpuk dengan rapi. Kalau anak buahnya menaruh dokumen tidak di tempatnya dia akan marah-marah. Buku-buku yang ada di rak tersusun rapi sesuai dengan yang paling besar sampai yang kecil. 

jadwal-sholat